The cost of publication in Journal of Biomedical Science is borne by the National Science Council, Taiwan.
Research
ZAK negatively regulates RhoGDIβ-induced Rac1-mediated hypertrophic growth and cell migration
-
* Corresponding author: Jaw-Ji Yang jjyang@csmu.edu.tw
- † Equal contributors
1 Graduate Institute of Chinese Medical Science, China Medical University, Taichung 404, Taiwan
2 Graduate Institute of Basic Medical Science, China Medical University, Taichung 404, Taiwan
3 Department of Health and Nutrition Biotechnology, Asia University, Taichung 413, Taiwan
4 School of Dentistry, Chung-Shan Medical University, Taichung 402, Taiwan
5 Institute of Medicine, Chung-Shan Medical University, Taichung 402, Taiwan
6 Chung-Shan Medical University Hospital, Chung-Shan Medical University, Taichung 402, Taiwan
7 Department of Orthopedics Surgery, Chung-Shan Medical University, Taichung 402, Taiwan
8 Midwestern University Chicago College of Osteopathic Medicine, Chicago, USA
9 Cardiovascular Center, Veterans General Hospital, Taichung 407, Taiwan
Journal of Biomedical Science 2009, 16:56 doi:10.1186/1423-0127-16-56
Published: 18 June 2009Additional files
Additional file 1:
Figure S1. The growth rate of H9c2 cells expressing RhoGDIβ and Rac1N17 grown in 10% fetal bovine serum with doxycycline.
Format: TIFF Size: 75KB Download file
Additional file 2:
Figure S2. Expression levels of cyclin-dependent kinase inhibitors p21 and p27 in H9c2 RhoGDIβ-expressing cells and H9c2 cells expressing both RhoGDIβ and Rac1N17.
Format: TIFF Size: 78KB Download file
Additional file 3:
Figure S3. SiRNA knockdown of RhoGDIβ inhibited cell migration of H9c2 RhoGDIβ-expressing cells.
Format: TIFF Size: 128KB Download file
Additional file 4:
Figure S4. Confocal microscopy of H9c2 and H9c2 cells expressing the indicated proteins.
Format: TIFF Size: 567KB Download file